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Nutrition: Antidoxidants

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Acute effects of strength exercise with blood flow restriction on vascular function of young healthy males
Conclusions: The results of this study suggest that one session of low intensity strength training with blood flow restriction does not reduce bioavailability of nitric oxide or induce redox imbalance in healthy young men.
Source: Jornal Vascular Brasileiro - July 10, 2018 Category: Surgery Source Type: research

Effects of Gentiana lutea root on vascular diseases.
CONCLUSION: Gentiana species and their constituents affect many different factors related to vascular disease development and progression. Therefore, Gentiana-based therapeutics represent potentially useful drugs for the management of vascular diseases. PMID: 32469702 [PubMed - as supplied by publisher]
Source: Current Vascular Pharmacology - May 28, 2020 Category: Drugs & Pharmacology Authors: Joksic G, Radak D, Sudar-Milovanovic E, Obradovic M, Radovanovic J, Isenovic ER Tags: Curr Vasc Pharmacol Source Type: research

Long-term dietary n3 fatty acid prevents aging-related cardiac diastolic and vascular dysfunction
CONCLUSIONS: We provide evidence and a simplified mechanistic insight on how long-term ALA supplementation is a successful strategy to prevent the development of age-related diastolic and vascular dysfunction.PMID:37105373 | DOI:10.1016/j.vph.2023.107175
Source: Vascular Pharmacology - April 27, 2023 Category: Drugs & Pharmacology Authors: Seyed Soheil Saeedi Saravi Nicole R Bonetti Ana Vukolic Daria Vdovenko Pratintip Lee Luca Liberale Cristina Basso Stefania Rizzo Alexander Akhmedov Thomas F L üscher Giovanni G Camici J ürg H Beer Source Type: research

VEGFR (Vascular Endothelial Growth Factor Receptor) Inhibition Induces Cardiovascular Damage via Redox-Sensitive ProcessesNovelty and Significance Oxidative Stress
Although VEGF (vascular endothelial growth factor) inhibitors (VEGFIs), are effective anticancer therapies, they cause hypertension through unknown mechanisms. We questioned whether changes in vascular redox state may be important, because VEGF signaling involves nitric oxide (NO) and reactive oxygen species. Molecular mechanisms, including NOS, NADPH oxidase (Nox)–derived reactive oxygen species, antioxidant systems, and vasoconstrictor signaling pathways, were probed in human endothelial cells and vascular smooth muscle exposed to vatalanib, a VEGFI. Vascular functional effects of VEGFI were assessed ex vivo in mouse a...
Source: Hypertension - March 7, 2018 Category: Cardiology Authors: Karla B. Neves, Francisco J. Rios, Lucas van der Mey, Rheure Alves-Lopes, Alan C. Cameron, Massimo Volpe, Augusto C. Montezano, Carmine Savoia, Rhian M. Touyz Tags: Basic Science Research, Oxidant Stress, Vascular Biology Original Articles Source Type: research

Angiotensin II and vascular damage in hypertension: Role of oxidative stress and sympathetic activation.
Abstract Reactive oxygen species (ROS) are oxygen derivates and play an active role in vascular biology. These compounds are generated within the vascular wall, at the level of endothelial and vascular smooth muscle cells, as well as by adventitial fibroblasts. Physiologically, ROS generation is counteracted effectively by the rate of elimination. In hypertension, a ROS excess occurs, which is not counterbalanced by the endogenous antioxidant mechanisms, leading to a state of oxidative stress. Angiotensin II, the active peptide of the renin-angiotensin-system (RAS), is a significant stimulus for ROS generation wit...
Source: Vascular Pharmacology - January 29, 2019 Category: Drugs & Pharmacology Authors: Masi S, Uliana M, Virdis A Tags: Vascul Pharmacol Source Type: research

Activation of M3AchR (Type 3 Muscarinic Acetylcholine Receptor) and Nrf2 (Nuclear Factor Erythroid 2-Related Factor 2) Signaling by Choline Alleviates Vascular Smooth Muscle Cell Phenotypic Switching and Vascular Remodeling.
CONCLUSIONS: These results reveal that choline impedes VSMC phenotypic switching, proliferation, migration, and vascular remodeling by activating M3AChR and Nrf2-antioxidant signaling and suggest a novel role for Nrf2 in VSMC phenotypic modulation. PMID: 32938216 [PubMed - as supplied by publisher]
Source: Arteriosclerosis, Thrombosis and Vascular Biology - September 16, 2020 Category: Cardiology Authors: He X, Deng J, Yu XJ, Yang S, Yang Y, Zang WJ Tags: Arterioscler Thromb Vasc Biol Source Type: research

Clock Gene Bmal1 Disruption in Vascular Smooth Muscle Cells Worsens Carotid Atherosclerotic Lesions
CONCLUSIONS: Bmal1 disruption in VSMCs worsens atherosclerosis by promoting VSMC migration and monocyte transmigration and impairing antioxidant function. Therefore, Bmal1 may be a potential therapeutic target and biomarker of atherosclerosis in the future.PMID:35236106 | DOI:10.1161/ATVBAHA.121.316480
Source: Arteriosclerosis, Thrombosis and Vascular Biology - March 3, 2022 Category: Cardiology Authors: Changpo Lin Lirong Xu Xiao Tang Xiaobo Li Chao Lu Qianyun Cheng Junhao Jiang Yang Shen Dong Yan Ruizhe Qian Weiguo Fu Daqiao Guo Source Type: research